文献管理-EndNote X7 文献の取込み

Home » 8000. 文献 » 文献管理-EndNote X7 文献の取込み
2017-05-15 8000. 文献 コメントはまだありません

文献管理-EndNote X7 文献の取込み

1 はじめに

EndNote X7 を使って文献の取込みを行うための手順を以下に記す。
取込み方法としては、2つの方法がある。一つ目は、EndNote から直接取込む方法、二つ目はブラウザを使って取込む方法である。ここでは、その両方の手順について記す。いずれの方法でもここでは、pubmed を例に説明する。

2 EndNote X7 から直接文献を取込む方法

2.1 文献の検索

  • EndNote X7 の左側のペインにある、「Online Search」の「PubMed」を選択する。
  • 検索パネルを表示させ、検索条件に該当するキーワードを入力する。
  • 「Search」ボタンを押す。
  • 検索結果の確認画面が表示されるので、よければ「OK」ボタンを押す。

 
0020.jpg

2.2 文献の検索結果

  • 検索結果が表示される。
  • 一通り検索結果で表示された文献を確認する。

 
0021.jpg
 

  • 取込み用のグループを作成する
  • [Groups]-[Create Group] により取込み用のグループを作成する。
  • グループが作成できたら、ドラッグアンドドロップにより、作成したグループに移す。

 
0022.jpg
 

3 Web サイト から EndNote X7 に文献を取込む

3.1 PubMed へアクセス

PubMed へアクセスしたら、検索窓にキーワードを入力し、「Search」ボタンを押す。
 
0023.jpg
 

3.2 検索結果の表示と取込み

 
0024.jpg
 

  • 該当する文献が見つかったら、「Send to」をクリックする。
  • 次に「File」を選択した後、「MEDLINE」を選択する。
  • 「Create File」ボタンを押す。

-「pubmed-result.txt」というファイル名が表示されるので「OK」ボタンを押す。
 
0025.jpg
 
「pubmed-result.txt」を開くと以下の様な内容が表示される。
 

PMID- 28077315
OWN – NLM
STAT- In-Data-Review
DA – 20170112
LR – 20170208
IS – 1872-7549 (Electronic)
IS – 0166-4328 (Linking)
VI – 322
IP – Pt A
DP – 2017 Mar 30
TI – Inhibition of iNOS alleviates cognitive deficits and depression in diabetic mice
through downregulating the NO/sGC/cGMP/PKG signal pathway.
PG – 70-82
LID – S0166-4328(16)31075-0 [pii] LID – 10.1016/j.bbr.2016.12.046 [doi] AB – Diabetes mellitus often results in a number of complications involving impaired
brain function, including cognitive deficits and depression. However, the
potential mechanisms for diabetes-related cognitive deficits and depression are
not fully understood. Neurons in the hippocampal, cortical and amygdala
functional regions are more susceptible to damage during hyperglycemia.
Neuroprotection in the brain can rescue cognitive deficits and depression induced
by hyperglycemia. This study investigated the potential mechanisms underlying
diabetes-related congnitive deficits and depression, determined whether the
inflammatory factor inducible nitric oxide synthase (iNOS) and the nitric oxide
(NO)/soluble guanylyl cyclases (sGC)/cyclic guanosine monophosphate
(cGMP)/protein kinase G (PKG) pathway, play key roles in cognitive deficits and
depression associated. In the present study, diabetic animal models were induced
by streptozotocin (STZ, 150mg/kg) in mice, and aminoguanidine (AG), a selective
inhibitor of iNOS, was given by intraperitoneal injection for 10 weeks. Blood
glucose, activities of NOS and the levels of NO in serum and brain regions were
measured. The spatial memory was detected using the Morris water maze test,
depressive behavior was evaluated by the tail suspension test (TST), forced
swimming test (FST), closed field test (CFT) and open field test (OFT). We also
detected neuronal survival and cleaved caspase-3 positive ratios in three brain
regions and the levels of iNOS, sGC, cGMP and PKG in hippocampus and frontal
cortex. Data indicated that diabetic mice exerted impairments in spatial memory,
decreased locomotor activity and increased immobile time in diabetic mice. In
addition, diabetic mice had significantly decreased surviving neuronal density
and showed signs of obvious neuronal injury in the hippocampus, frontal cortex
and amygdala. iNOS overexpression and its associated signaling pathway
NO/sGC/cGMP/PKG in the hippocampus and frontal cortex were implicated during
hyperglycemia. However, AG improved the behavior disorders, reduced the activity
of iNOS, protected nerve cells and inhibited the level of iNOS, sGC, PKG and
cleaved caspase-3 in the hippocampus and cortex. These results suggested that
iNOS/NO/sGC/cGMP/PKG signal pathway is a key feature of cognitive deficits and
depression associated with diabetes. AG ameliorated cognitive deficits and
depression in diabetic mice by exerting anti-inflammatory and neuroprotective
effects by suppressing iNOS-associated signaling pathways.
CI – Copyright (c) 2017 Elsevier B.V. All rights reserved.
FAU – Zhou, Xiao Yan
AU – Zhou XY
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Zhang, Fang
AU – Zhang F
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Ying, Chang Jiang
AU – Ying CJ
AD – Department of Endocrinology, Affiliated Hospital of Xuzhou Medical University,
Xuzhou, Jiangsu, 221002, PR China.
FAU – Chen, Jing
AU – Chen J
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Chen, Ling
AU – Chen L
AD – The Graduate School, Xuzhou Medical University, Xuzhou, Jiangsu, 221002, PR
China.
FAU – Dong, Jing
AU – Dong J
AD – The Graduate School, Xuzhou Medical University, Xuzhou, Jiangsu, 221002, PR
China.
FAU – Shi, Yue
AU – Shi Y
AD – The Graduate School, Xuzhou Medical University, Xuzhou, Jiangsu, 221002, PR
China.
FAU – Tang, Mang
AU – Tang M
AD – The Graduate School, Xuzhou Medical University, Xuzhou, Jiangsu, 221002, PR
China.
FAU – Hu, Xiao Tong
AU – Hu XT
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Pan, Zhi Hua
AU – Pan ZH
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Xu, Na Na
AU – Xu NN
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China.
FAU – Zheng, Kui Yang
AU – Zheng KY
AD – Department of Pathogen Biology and Immunology, Laboratory of Infection and
Immunity, Xuzhou Medical College, Xuzhou, Jiangsu, 221004, PR China.
FAU – Tang, Ren Xian
AU – Tang RX
AD – Laboratory of Morphology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China; Department of Pathogen Biology and Immunology, Laboratory of Infection and
Immunity, Xuzhou Medical College, Xuzhou, Jiangsu, 221004, PR China. Electronic
address: tangrenxian-t@163.com.
FAU – Song, Yuan Jian
AU – Song YJ
AD – Jiangsu Key Laboratory of Brain Disease Bioinformation, Xuzhou Medical
University, Xuzhou, Jiangsu, 221004, PR China; Department of Genetics, Research
Center for Neurobiology, Xuzhou Medical University, Xuzhou, Jiangsu, 221004, PR
China. Electronic address: biosongyuanjian@126.com.
LA – eng
PT – Journal Article
DEP – 20170108
PL – Netherlands
TA – Behav Brain Res
JT – Behavioural brain research
JID – 8004872
OTO – NOTNLM
OT – Aminoguanidine
OT – Cognitive deficits
OT – Depression
OT – Nitric oxide
OT – iNOS
EDAT- 2017/01/13 06:00
MHDA- 2017/01/13 06:00
CRDT- 2017/01/13 06:00
PHST- 2016/11/15 [received] PHST- 2016/12/30 [revised] PHST- 2016/12/31 [accepted] AID – S0166-4328(16)31075-0 [pii] AID – 10.1016/j.bbr.2016.12.046 [doi] PST – ppublish
SO – Behav Brain Res. 2017 Mar 30;322(Pt A):70-82. doi: 10.1016/j.bbr.2016.12.046.
Epub 2017 Jan 8.

 

3.3 EndNote X7 への取込み

ダウンロードしたテキストファイルからEndNote X7 へ文献情報を取込む

  • EndNote X7 の[ファイル]-[インポート]-[ファイル]よりダウンロードしたファイルを指定する。

 
0026.jpg
 

4 引用文献

讃岐 美智義. (2015). 最新 EndNote 活用ガイド デジタル文献整理術 (Vol. 第6版).

コメントを残す

メールアドレスが公開されることはありません。 * が付いている欄は必須項目です